|
|
1391/11/29، جلد ۱۲، شماره ۱، صفحات -
|
|
|
| عنوان فارسی |
|
|
| چکیده فارسی مقاله |
|
|
| کلیدواژههای فارسی مقاله |
|
|
| عنوان انگلیسی |
Asthma and Influenza Virus Infection: Focusing on Cell Death and Stress Pathways in Influenza Virus Replication |
|
| چکیده انگلیسی مقاله |
Asthma is one the fastest growing syndromes in many countries and is adding a huge cost to the health care system. Increasing reports have linked airway infectious diseases to asthma. Influenza is one the most serious airway infectious diseases and in recent years there have been some serious influenza virus pandemics which caused increased fatality in numerous different populations. Diverse host response pathways during virus infection have been identified, including different cell death and survival pathways. These pathways include 1)programmed cell death I (apoptosis), 2) programmed cell death II (autophagy), and 3) endoplasmic reticulum stress with subsequent unfolded protein (UPR) response. There has been extensive research on the regulatory roles of these pathways during the influenza virus life cycle. These studies address the benefits of enhancing or inhibiting these pathways on viral replication. Here we review the most recent and significant knowledge in this area for possible benefits to clinicians and basic scientist researchers in different areas of the respiratory and virology sciences. Keywords: Apoptosis, Autophagy; Endoplasmic Reticulum Stress; Unfolded Protein Response; Virus-induced autophagy; Virus-induced apoptosis. |
|
| کلیدواژههای انگلیسی مقاله |
Apoptosis, Autophagy; Endoplasmic Reticulum Stress; Unfolded Protein |
|
| نویسندگان مقاله |
42389---42390---42391---42392---42393---42394---42395---42396--- |
|
| نشانی اینترنتی |
http://ijaai.tums.ac.ir/index.php/ijaai/article/viewArticle/688 |
| فایل مقاله |
فایلی برای مقاله ذخیره نشده است |
| کد مقاله (doi) |
|
| زبان مقاله منتشر شده |
en |
| موضوعات مقاله منتشر شده |
|
| نوع مقاله منتشر شده |
Articles |
|
|
|
برگشت به:
صفحه اول پایگاه |
نسخه مرتبط |
نشریه مرتبط |
فهرست نشریات
|
